← All studies

1995 · Gillespie — Energetics of Molecular Hydrogen Oxidation in the Oral Pathogen Campylobacter rectus

Original title: Energetics of molecular hydrogen oxidation in the oral pathogen Campylobacter rectus.

Super-Abstract

This in-vitro study examined how the oral pathogen Campylobacter rectus oxidizes molecular hydrogen (H₂) as an energy source — a fundamental microbiology question about bacterial hydrogen metabolism in the oral cavity. No abstract is publicly available; details are accessible via the DOI. (Clinical Infectious Diseases, 1995.)

Classified as a Mechanism / Preclinical study using Drinking (HRW). See Methodology for how we grade evidence.

Commentary

This study investigates the bioenergetics of H₂ oxidation in Campylobacter rectus — a gram-negative bacterium associated with periodontal disease. The relevance to „hydrogen medicine” lies in understanding how certain oral bacteria use H₂ as an electron donor. This is basic microbiology, not a therapeutic study. No abstract is available in the database entry; a full assessment requires access to the original publication (DOI: 10.1093/clinids/20.supplement_2.s172). Results should not be extrapolated to therapeutic H₂ in humans.

Key quotes

  1. „Energetics of molecular hydrogen oxidation in the oral pathogen Campylobacter rectus.“ — title only — no abstract available; all quotes are drawn from the title

Our assessment

This is an in-vitro microbiology study on bacterial H₂ metabolism — not a therapeutic hydrogen study. Because no abstract is available in the record, a full evaluation is not possible here. This is not evidence for any human health effect of H₂. The study is of basic scientific interest for understanding bacterial energy metabolism in the oral microbiome. Readers seeking clinical evidence on H₂ therapy should consult the original paper directly via its DOI.

Study design

Source & links

Screenshot of the PubMed page

Screenshot — PubMed 7548544

This page mirrors the published abstract (© the authors / publisher) for reference and citation. The canonical source is the PubMed record linked above. This is not medical advice.